This Article Full Text (PDF) References Alert me when this article is cited Alert me if a correction is posted Citation Map Services Email this article to a friend Similar articles in this journal Similar articles in PubMed Alert me to new issues of the journal Add to Saved Citations Download to citation manager Request Permissions Request Reprints Add to My Marked Citations Citing Articles Citing Articles via HighWire Citing Articles via Google Scholar Citing Articles via Scopus Google Scholar Articles by Smith, C. J. Articles by Ryan, B. A. Search for Related Content PubMed PubMed Citation Articles by Smith, C. J. Articles by Ryan, B. A. Pubmed/NCBI databases Substance via MeSH Medline Plus Health Information Indoor Air Pollution Social Bookmarking                   What's this?

Cardiovascular effects of odors

Research and Development, R.J. Reynolds Tobacco Company, Winston-Salem, North Carolina

Sandra M. Scott

Research and Development, R.J. Reynolds Tobacco Company, Winston-Salem, North Carolina

Brett A. Ryan

Research and Development, R.J. Reynolds Tobacco Company, Winston-Salem, North Carolina

Several occupational and residential settings can expose both normal and sensitive human subjects to odors and irritants. These settings include intensive agricultural operations housing swine and poultry, cigarette-smoke-filled bars, landfills and manufacturing processes. The literature suggests that adverse sensory reactions to strong odors and irritants may lead to the release of catecholamines and stress hormones. Physiological and biochemical measurements related to cardiovascular risk, e.g., blood pressure, heart rate, high-density lipoprotein (HDL) cholesterol level and serum triglyceride level, may be altered as a result of exposure to odor and irritant-induced release of catecholamines. Further work in the form of field studies and chamber exposure protocols is required to determine whether the physiological and biochemical changes observed to date represent an increase in cardiovascular risk, or are reversible changes within the normal homeostatic range.

Key Words: cardiovascular effect • irritant • odor

Toxicology and Industrial Health, Vol. 15, No. 7, 595-601 (1999)
DOI: 10.1177/074823379901500701


CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    What's this?

This article has been cited by other articles:

				D. Oakes, H. Ritchie, P. Woodman, E Narup, M Moscova, K Picker, and W. Webster Genotoxicity studies of a desealant solvent mixture, SR-51(R) Toxicology and Industrial Health, February 1, 2009; 25(1): 5 - 13. [Abstract] [PDF]

				J.-F. Argacha, D. Adamopoulos, M. Gujic, D. Fontaine, N. Amyai, G. Berkenboom, and P. van de Borne Acute Effects of Passive Smoking on Peripheral Vascular Function Hypertension, June 1, 2008; 51(6): 1506 - 1511. [Abstract] [Full Text] [PDF]

				E. K. Tong and S. A. Glantz Tobacco Industry Efforts Undermining Evidence Linking Secondhand Smoke With Cardiovascular Disease Circulation, October 16, 2007; 116(16): 1845 - 1854. [Abstract] [Full Text] [PDF]

				T. Raupach, K. Schafer, S. Konstantinides, and S. Andreas Secondhand smoke as an acute threat for the cardiovascular system: a change in paradigm Eur. Heart J., February 2, 2006; 27(4): 386 - 392. [Abstract] [Full Text] [PDF]

				N. Fiedler, N. Giardino, B. Natelson, J. E. Ottenweller, C. Weisel, P. Lioy, P. Lehrer, P. Ohman-Strickland, K. Kelly-McNeil, and H. Kipen Responses to Controlled Diesel Vapor Exposure Among Chemically Sensitive Gulf War Veterans Psychosom Med, July 1, 2004; 66(4): 588 - 598. [Abstract] [Full Text] [PDF]

				M. G. Warner Complementary and Alternative Therapies for Hypertension Complementary Health Practice Review, October 1, 2000; 6(1): 11 - 19. [PDF]